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Cancers Packages & Protocols Spotlight Products
Educational Products Personal Care Protocol Instructions
Low pH?
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Stomach mender is designed to:
I have spoken to natural practitioners that claim that 90 percent of their practice is based on people misapplication of basic health, how the absorption of nutrition is critical to health. Said simply one can eat the very best organic food and supplements on the planet, and still remain sick if the food passes through the body and does not uptake into the body. If the food leaves the body unassimilated then the nutritional content was wasted. Proprietary Blend: Chaga Extract Ox bile Pepsin This product is pure nutrition; no fillers, additives or synthetic chemicals. The gelatin capsule complies with the requirements published in: The United States Pharmacopoeia (USP); XXIV / National Formulary (NF) 19 Kosher and Halal certified First Day Take one 1 capsule Stomach Mender 5 minutes before each meal. Take one 1 capsule Stomach Mender 5 minutes before each meal.
Evaluate the color and consistency of your stool. If your stool is not a dark color increase your dosage as follows: Take 2 capsules Stomach Mender 5 minutes before each meal. Take 2 capsules Stomach Mender 5 minutes before each meal. Take 2 capsules Stomach Mender 5 minutes before each meal. Third Day Evaluate the color and consistency of your stool. If your stool is not a dark color increase your dosage as follows: Take 3 capsules Stomach Mender 5 minutes before each meal. Take 3 capsules Stomach Mender 5 minutes before each meal. Take 3 capsules Stomach Mender 5 minutes before each meal. Evaluate the color and consistency of your stool. Keep increasing until the color of the stool is corrected. Lower your dosage when your stool becomes a dark brown and it sinks through the water and it sticks to side of the bowl (sticking to the bowl means you've taken too much). How to Lower Dosage... Example, if you were taking the following when your stool became corrected: Adjust to: Or: Explanation Make sure you digest all your food. However, there is no reason to waste the product. What is needed is for all your food to be properly digested. This can open your bile ducts and help the body uptake essential oils, nourishing the cells. This process will also open up the biliary ducts, allowing gall stones to naturally flow through. Upset stomach, gas, diarrhea if the product is not balanced in the body.
Discomfort 0-2 Hours After EatingThe health transit time out of the stomach is about 2 hours. With that concept in mind if you experience any of the following symptoms you might not be breaking down your food properly:
The rhythmical contraction of the intestines, that crush and move food through the GI track, called peristalsis, is triggered by the stomach acid.
Function of Pepsin The enzyme pepsin is secreted in inactive form called pepsinogen. Proper functioning pepsinogen is formed into pepsin, when the pH is correct in the stomach. The ideal pH of the stomach is between 1.5 -3.5 this pH corresponds the activation range of the human stomach. Gastric acids are secreted into the stomach commonly known as Hydrochloric acid but this is just one the types of acids. Gastric acids typical pH of 1 to 2 and is composed of hydrochloric acid (HCl) around 0.5%, and large quantities of potassium chloride (KCl) and sodium chloride (NaCl).The pH of the stomach environment is generally around 1.5 to 3.5, which as mentioned, is necessary for the activation and optimal activity of pepsin. Function of Bile
Bile acids are polyhydroxylated steroidal acids obtained from the digestive systems of vertebrates. The most abundant mammalian bile acids are hydroxy derivatives of cholanoic acid . The human bile acid pool consists mainly (~ 90%) of cholic acid, chenodeoxycholic acid, and deoxycholic acid. Cholic acid and chenodeoxycholic acid are synthesized from cholesterol in the liver via many enzymatic steps. These primary bile acids are conjugated via amide bond to glycine or taurine to give bile salts such as glycocholate and taurocholate before secretion into the bile. Bile is stored in the gallbladder and emptied after food intake into the small intestine where bile acids aid in the digestion and resorption of lipids and lipid-soluble vitamins . A part of the primary bile acids is transformed into the secondary bile acids, deoxycholic acid and lithocholic acid in the colon. After their absorption in the small intestine and colon, bile acids gain entry to the liver in order to begin their enterohepatic circulation once again. Hofmann has widely considered the role of bile acids in metabolism, their impact on plasma cholesterol levels, and diseases caused by defects in bile acid metabolism by Jari Tamminen et al 2001. The primary function of bile salts is to emulsify the fat one consumes. This is simple the action of a detergent. Bile functionally acts as soap inside the human body braking up fat globules into small components lipase can break down. Unless one has a clogged bile duct one does not think of the importance of this process. But one should consider the action, if one does not have good health. Bile salts influence the absorption of dietary fat has been known for many years, adult humans produce a daily average of 500 ml of bile, per day. Bile Salts are a complex fluid containing, cholesterol, bilirubin and other organic molucules, Hofmann 1998. Bile salts aid in the digestion and absorption of fats and fat-soluble vitamins in the small intestine. The most abundant bile salts in humans are cholate and deoxycholate. Bile is stored and concentrated in the gallbladder during fasting and in released through the bile duct into the intestine during food passage. Because bile salts are detergents, bile possesses strong antimicrobial activity. In 1897, in an elegant and seldom cited paper, Moore and Rockwood reported that active fat absorption could be observed from a loop of intestine whose contents did not appear to be emulsion. These workers also demonstrated that bile salt solutions could dissolve large amounts of fatty acids, and they were quite puzzled by this phenomenon. It is now recognized that the ability of bile salt solutions to dissolve lipids is merely a reflection of their detergent properties. Above a critical concentration, bile salt molecules aggregate to form micelles; such micellar aggregation is a characteristic of all detergents. All micellar solutions dissolve water insoluble materials to some extent, as the inside of the micelle can be considered as a separate, nonaqueous phase with distinctive solvent properties. Fatty acids and monoglycerides possess a remarkable solubility in bile salt solutions. We have recently presented preliminary evidence for these lipids being present in micellar solution during fat digestion and absorption. We now describe experiments in detail in which we have collected human intestinal content during fat digestion and absorption and have separated it by ultracentrifugation at 370 C into an infranatant micellar phase and a supernatant oil phase. Inflammatory Bowel Disease An extract of Chaga reduced the oxidative stress on lymphocytes from patients with inflammatory bowel disease. A hot water extract of Chaga exhibited inhibitory and proapoptotic actions against colon cancer cell proliferation via up-regulation of Bax and caspase-3 and down-regulation of Bcl-2 Najafzadeh et al. theorized that Ulcerative colitis (UC) and Crohn’s disease (CD) are inflammatory disorders of the gastrointestinal tract, which are unevenly distributed within the populations throughout the world. Najafzadeh explains that although the exact cause of inflammatory bowel disease (IBD) remains unknown, the epidemiology of inflammatory bowel disease has provided an insight into the pathogenesis of the disease by examining geographic, ethnic and other inflammatory bowel disease risk factors (genetic, environmental, etc.) as well as their natural history (12). Interestingly, reactive oxygen species (ROS) also know as free radicals (13) are produced in abnormally high levels in cells from of inflammatory bowel disease patients leading to oxidative stress/ free radicals damage and thus to DNA damage due to an imbalance between innate and exogenous antioxidants and reactive oxygen species. Oxidative stress has been linked to cancer, ageing, atherosclerosis, ischaemic injury, inflammation, and neurodegenerative diseases. Medically, inflammatory bowel disease is characterized by the infiltration of CD4þ T-lymphocytes and other mononuclear cells into inflamed mucosal regions. During this process, interleukin (IL)-16 exerts a strong chemo-attractant activity towards CD4þ cells. Moreover, IL-16 activates the expression and production of pro-inflammatory cytokines such as IL-1 beta, IL-6, IL-15 and tumor necrosis factor alpha (TNF-alpha) in human monocytes. In 2007, Yuki Nakajima et al. studied Chaga extract from water also know as a decoction. The antioxidant properties compared to other medical mushrooms were compared. When compared to Almond Mushroom/Agaricus blazei mycelia, Reishi/Ganoderma lucidum, and Meshimakobu/Phellinus linteus, Chaga demonstrated the strongest antioxidant activity among the mushrooms examined in terms of both superoxide and hyproxyl radicals scavenging activates. Investigation determined whole plant Chaga decoction superior to other mushrooms examined for the antioxidant properties compared to the mushrooms examined in this study.
Human Digestive Substances The human digestive system contains copious enzymes to help digest our food and fight disease: Potential Disease State: If you have stomach ulcer most likely you are infected with the bacteria H. pylori (Helicobacter pylori). H. pylori damages the mucosa lining of the stomach. H. pylori in the stomach causes chronic, low-level inflammation and is linked to gastric ulcers and stomach cancer, how does it do this? H. pylori must cope with highly acidic conditions in which other bacteria are unable to survive. It is well known however, that the bacterium accomplishes this by producing ammonia to neutralize the acid in its surroundings area. The pH change allows H. pylori two swim across and through its mucin barrier infecting the stomach with the bacterium, establish colonies, and attack surface cells forming ulcers.
Gastric mucosa know as mucin is a protein: In the presence of acid mucin forms a protective gel, which acts as a physical barrier that stops harmful bacteria from reaching the cell wall. Gastric mucosas are produced by goblet cells whose sole function is to secrete mucin, which when dissolved in water form mucus. One of the stomachs functions is to destroy bacteria and viruses using its highly acidic environment. This acid also has the ability to damage the lining of the stomach this is where the mucosa lining of the stomach is so important. The action of the stomach is by secreting mucin and bicarbonate via its mucous cells, the outer end of mucosa where the food is found has a pH around 1-1.5 and as we get closer to the lining of the stomach the pH is closer to seven (7) that is to say neutral. There are three types of glands cardiac glands, oxyntic glands, Pyloric glands: Cardiac glands (in the proximal part of the stomach), The cardiac glands mainly contain mucus producing cells. Oxyntic glands produce pepsin, The bottom part of the oxyntic glands is dominated by zymogen cells that produce pepsinogen. Pyloric glands. The pyloric glands contain mucus-secreting cells. Several types of endocrine cells are found in all regions of the gastric mucosa. In the pyloric glands contain gastrin producing G cells; this hormone stimulates acid production from the parietal cells. The A cells produce glucagon, which mobilizes the hepatic glycogen, and the enterochromaffin cells that produce serotonin, which stimulates the contraction of the smooth muscles. Parietal cells, which secrete hydrochloric acid are scattered in the glands, with most of them in the middle part. The upper part of the glands consist of mucous neck cells; in this part the dividing cells are seen. ECL (enterochromaffine-like) cells, found in the oxyntic glands release histamine, which also is a powerful stimulant of the acid secretion.
Pancreatic juice, composed of the secretions of both ductal and acinar cells, is made up of the following digestive enzymes:
The enzyme pepsin functions to break proteins into smaller polypeptides. Stool Health Question We All Need to ConsiderAlternative practitioners often ask patients about their stool as part of their health assessment. Find out what normal stool should look like, and learn about the causes of green stool, pale stool, and yellow stool, blood in stool, mucus in stool, pencil thin stool, infrequent stool, and what to do about it. What Does an Ideal Bowel Movement Look Like?Your stool color can vary greatly from day to day. All shades of brown are considered normal, but medium to dark brown are desirable. Your stool should leave the body easily without pain, straining or any discomfort. Your stool should have the consistency of paste, e.g. toothpaste.
Black Stool
*Bleeding in the stomach or the intestines can change the color of stool. If bleeding occurs in the stomach or the upper part of the small intestine, the stool may turn black and tacky, described medically as black, tarry stool. Generally, black, tarry stool also is foul-smelling. If the stool color is dark because of any of these medications, it is typically not tacky / sticky in texture and is not foul-smelling. **Anthocyanins are water-soluble pigments that may appear red, purple, or dark blue according to pH. They belong to a parent class of molecules called flavonoids. So is you are juicing and your stool is changing color it could be the flavonoids found in your food.Red stool;
Tomato Soup or Juice, Large Amounts Beets Anthocyanin* Anthocyanins are water-soluble pigments that may appear red, purple, or dark blue according to pH. They belong to a parent class of molecules called flavonoids. So is you are juicing and your stool is changing color it could be the flavonoids found in your food. Stool That Sinks Quickly Rapidly sinking dense stool, when it hits the water splashes up can indicate that a person might not be eating enough fiber-rich foods, also dehydration* can be a concern. Stool can be dark if it’s been sitting in the intestines for a prolonged period of time over 24 hours. One should have a minimum of 1bowl movement per day, if not consider** Pale Gray Stool
Stool that is pale or grey may be caused by insufficient bile output due to conditions such as:
Disease and disorders can hinder the productions and secretion of bile acids into the duodenum. After secreted into the duodenum the pH is raised allowing the pancreatic enzyme lipase to interact with bile. Bile acts like a detergent braking up globules of fat in manageable particle sizes allowing these essential oils to nourish the body. Pale Soft, foul-smelling stool that floats, sticks to the side of the bowl, or is difficult to flush away may mean there is increased undigested fat in the stools, medical known as steatorrhea / fat malabsorption. One needs to keep in mind fat absorption is interdependent upon bile and lipase. Bile acids are produced in the liver and stored and secreted by the gallbladder, lipase is producd by the pancreases., this is the normal intestine function. Absence of bile is often due to blockage of the biliary tract and can result in pale colored fatty stool and jaundice. Complete absence of pancreatic lipases is uncommon, but lipase deficiency, are more common as we age. The complete absence or non-production of lipase is rare, but can occur as a result of a diseased pancreas, cystic fibrosis, or an abnormality that is present at birth. Inflammation of the lining of the intestines, which may occur with conditions such as ulcerative colitis (inflammation of the colon and rectum), Crohn’s disease (inflammation of the bowels), and celiac disease (a severe sensitivity to gluten in the diet), can interfere with absorption of fats. If you are suffering with inflamed bowl, I have seen hundreds of people health with Chaga Extract. Green Stool
The liver constantly makes bile, a bright green fluid, which is secreted directly into the gallbladder. Bile acids are held until the food is released from the stomach, then travels into the duodenum/small intestine.
The duodenum wall is composed of a very thin layer of cells that form the mucous membrane. Yellow Stool Yellow-green fluid that digests fats in your stool. As bile acids travel through your gastrointestinal tract, they are chemically altered by enzymes, which change the pigments from green to shades of yellow/brown to brown/dark brown depending upon the concentration of bile acids and enzymes present in stool. Yellow Stool GERD
Malabsorption Disorders Malabsorption is a state arising from abnormality in absorption of food nutrients across the gastrointestinal (GI) tract. Impairment can be of single or multiple nutrients depending on the abnormality. This may lead to malnutrition and variety of anaemias.
Loose Stool:
Black Stool
*Bleeding in the stomach or the intestines can change the color of stool. If bleeding occurs in the stomach or the upper part of the small intestine, the stool may turn black and tacky, described medically as black, tarry stool. Generally, black, tarry stool also is foul-smelling. If the stool color is dark because of any of these medications, it is typically not tacky / sticky in texture and is not foul-smelling. **Anthocyanins are water-soluble pigments that may appear red, purple, or dark blue according to pH. They belong to a parent class of molecules called flavonoids. So is you are juicing and your stool is changing color it could be the flavonoids found in your food.Pencil Thin Stool Persistent pencil thin stool, which may be solid or loose, is one of the symptoms of colorectal polyps or cancer. As the size of the polyp or cancer increases, it can decrease the internal diameter of the colon or rectum so that only thin stool can pass through. Other symptoms can include blood in the stool, abdominal discomfort, and unintended weight loss, particularly in the case of advanced colorectal cancer. Colorectal cancers are diagnosed in about 140,000 people in the United States per year (Source: CDC).
Mucus in Stool Mucus in stool often indicates there is inflammation of the colon. Mucus in stool can occur with either constipation or diarrhea. It's usually appears whitish in color.
Symptoms and Corrections Bacterial overgrowth can be corrected by using friendly bacteria. In essence the friendly bacteria are an antagonist against unfriendly bacteria as the friendly bacteria colonies they overtake the unfreandly correcting the colon health. The majority of health bacteria reside in the large intestines. Common symptoms of unfriend bacteria are bloating and gas usually worsen after eating carbohydrate/sugar, the bacteria use this food souse to colonize the body, making one sicker. One point I believe we all need to understand is all carbohydrate are converted into sugar, and sugar is essential to human life. The fact is the brain exclusively feeds on sugar and the muscles need sugar as energy, the fact is, if you don’t consume significant amounts of sugar your body will convert fat and protein into glucose/sugar*.
People with food allergies and food sensitivities react to specific foods a good example would be a person with:
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Symptoms of Inflammatory Bowl Disease Ulcerative colitis, Crohn's disease, and celiac disease are often accompanied by diarrhea. Rectal bleeding can also occur with ulcerative colitis and Crohn's disease. Food sensitivities If the tongue has a thick coating with teethmarks on the sides, greasy foods, dairy products, and wheat may be contributing to the mucus in stool. Avoiding these foods is often recommended, at least until the condition improves.
Bacterial or parasitic infections can also cause mucus in stool. They are often accompanied by a sudden onset of diarrhea, lower abdominal cramping, urgency and possibly blood in the stools. Symptoms of Intestinal Parasites
Intestinal parasites can cause one or more of the following symptoms: Parasites Giardia lamblia Infection is transmitted by contaminated drinking water.
Bacterial enterocolitis *During protein metabolism or insignificant carbohydrate uptake, protein can be converted to glucose in a process called gluconeogenesis, the formation of glucose from non-carbohydrate sources. The first step in protein metabolism is to break it into its constituent amino acids. These are absorbed into the blood stream. The second step is to break down the amino acids into their constituent parts—catabolism, removes the nitrogen or amino group from the amino acids. The process is called deamination.
People can become easily infected by
Rotavirus is the most common cause of severe diarrhea among infants and young children, and is one of several viruses that cause infections often called stomach flu, despite having no relation to influenza. By the age of five, nearly every child in the world has been infected with rotavirus at least once. However, with each infection, immunity develops, and subsequent infections are less severe; adults are rarely affected. There are five species of this virus, referred to as A, B, C, D, and E. Rotavirus A, the most common, causes more than 90% of infections in humans. Sapoviruses are caliciviruses that together with the noroviruses, are the most common cause of acute viral gastroenteritis in adults. The type species is Sapporo virus and it is named after Sapporo, Japan where the virus was first discovered following an outbreak of gastroenteritis in an orphanage. Unlike norovirus, this virus generally only causes mild gastroenteritis in young children. A different type of virus called rotavirus is the main cause of severe, acute gastroenteritis in children under the age of five years. Bacterial Food Poising Common foodborne diseases Bacterial food poisoning caused by Salmonella species, Shigella species, Staphylococcus aureus, Clostridium perfringens, Bacillus cereus and certain strains of Escherichia coli also commonly causes episodic diarrhea. Campylobacter is a bacterial pathogen that causes fever, diarrhea, and abdominal cramps. It is the most commonly identified bacterial cause of diarrheal illness in the world. These bacteria live in the intestines of healthy birds, and most raw poultry meat has Campylobacter on it. Eating undercooked chicken, or other food that has been contaminated with juices dripping from raw chicken is the most frequent source of this infection. E. coli O157:H7 is a bacterial pathogen that has a reservoir in cattle and other similar animals. Human illness typically follows consumption of food or water that has been contaminated with microscopic amounts of cow feces. The illness it causes is often a severe and bloody diarrhea and painful abdominal cramps, without much fever. In 3% to 5% of cases, a complication called hemolytic uremic syndrome (HUS) can occur several weeks after the initial symptoms. This severe complication includes temporary anemia, profuse bleeding, and kidney failure. Salmonella is also a bacterium that is widespread in the intestines of birds, reptiles and mammals. It can spread to humans via a variety of different foods of animal origin. The illness it causes, salmonellosis, typically includes fever, diarrhea and abdominal cramps. In persons with poor underlying health or weakened immune systems, it can invade the bloodstream and cause life-threatening infections. Shigella is a genus of Gram-negative, nonspore forming, rod-shaped bacteria closely related to Escherichia coli and Salmonella. It is only naturally found in humans and apes Shigellosis is an infectious disease caused by a group of bacteria called. Most who are infected with Shigella develop diarrhea, fever, and stomach cramps starting a day or two after they are exposed to the bacteria. The diarrhea is often bloody. Shigellosis usually resolves in 5 to 7 days. Persons with shigellosis in the United States rarely require hospitalization. A severe infection with high fever may be associated with seizures in children less than 2 years old. Some persons who are infected may have no symptoms at all, but may still pass the Shigella bacteria to others. Viruses Food Poising Adenoviruses can also cause gastroenteritis (stomach flu). A combination of conjunctivitis and tonsilitis is particularly common with adenovirus infections. Astrovirus was first discovered after following an outbreak of diarrhea in humans. In addition to humans, astroviruses have now been isolated from numerous mammalian animal species (and are classified as genus Mammoastrovirus) and from avian species such as ducks, chickens, and turkey poults (classified as genus Aviastrovirus). Noroviruses, or Norwalk-like virus is an extremely common cause of foodborne illness, though it is rarely diagnosed, because the laboratory test is not widely available. It causes an acute gastrointestinal illness, usually with more vomiting than diarrhea, that resolves within two days. Unlike many foodborne pathogens that have animal reservoirs, it is believed that Norwalk-like viruses spread primarily from one infected person to another. Infected kitchen workers can contaminate a salad or sandwich as they prepare it, if they have the virus on their hands. Infected fishermen have contaminated oysters as they harvested them. Parasites Causes Sickness Cryptosporidiosis, also known as crypto, is a parasitic disease caused by Cryptosporidium, a protozoan parasite in the phylum Apicomplexa. It affects the intestines of mammals and is typically an acute short-term infection. It is spread through the fecal-oral route, often through contaminated water; the main symptom is self-limiting diarrhea in people with intact immune systems. Cyclospora is a parasite that is composed of one cell. It is too small to be seen without a microscope. Its full name is Cyclospora cayetanensis. Cyclospora infects the small intestine (bowel) and usually causes watery diarrhea, with frequent bowel movements. Other symptoms include loss of appetite, weight loss, bloating, increased gas, stomach cramps, nausea, vomiting, tiredness, muscle aches, and low-grade fever. Other infectious organisms can cause similar illness. Some persons infected with Cyclospora do not develop any symptoms. Without treatment, the infection can cause weeks of severe tiredness, loss of appetite, dehydration (loss of fluids), and weight loss. The first human cases were reported in 1979, but cases began being reported more often in the mid-1980s. This may be partly because of the availability of better tests to detect the parasite in stool specimens. Several recent, very large outbreaks linked to raspberries, mesclun lettuce, and possibly basil have increased concerns about Cyclospora and stimulated health officials to learn more about how it is transmitted and what factors contribute to its spread. Entamoeba histolytica is the third leading cause of morbidity and mortality due to parasitic disease in humans (after malaria and schistosomiasis) and is estimated to be responsible for between 50,000 and 100,000 deaths every year. Infection can lead to amoebic dysentery, resulting from trophozoites invading the intestinal wall, and amoebic liver abscess and other extraintestinal lesions, resulting from spread of trophozoites from the intestine via the bloodstream. Differentiation among species of Entamoeba is clearly important for accurate diagnosis of individuals at risk and to prevent unnecessary or inappropriate therapy. Giardia lamblia is a genus of protozoan parasites lives inside the intestines of infected humans or other animals. Individuals become infected through ingesting or coming into contact with contaminated food, soil, or water. The Giardia parasite originates from contaminated items and surfaces that have been tainted by the feces of an infected animal. The symptoms of Giardia causing giardiasis, commonly known as Beaver fever, which may begin to appear 1–2 weeks after infection, include diarrhea, excess gas, stomach or abdominal cramps, upset stomach, and nausea. In addition to disease caused by direct infection, some foodborne diseases are caused by the presence of a toxin in the food that was produced by a microbe in the food. For example, the bacterium Staphylococcus aureus can grow in some foods and produce a toxin that causes intense vomiting. The rare but deadly disease botulism occurs when the bacterium Clostridium botulinum grows and produces a powerful paralytic toxin in foods. These toxins can produce illness even if the microbes that produced them are no longer there.
Bowel Infections and Food Poisoning The most commonly recognized foodborne infections are those caused by the bacteria Campylobacter, Salmonella, and E. coli O157:H7, and by a group of viruses called calicivirus, also known as the Norwalk and Norwalk-like viruses. Infections bowel share common symptoms although caused by different organisms’ e.g. viruses, bacteria and certain parasites frequently cause loose stools. Viral gastroenteritis, an inflammation of the stomach and intestines, cause of episodic diarrhea. Infrequent StoolWith constipation, infrequent or hard stool is passed with straining. Learn about the causes of infrequent stool. Pellet StoolPellet stool is stool that comes out in small, round balls. In traditional Chinese medicine, pellet stool is caused by a condition known as liver qi stagnation. Liver qi stagnation can be brought on by stress. Lack of exercise can worsen the problem. The Pancreas The pancreas secretes a number of enzymes including maltase, sucrase, and lactase (which break down the corresponding disaccharides), trypsin and chymotrypsin (which further digest protein), and pancreatic lipase (which breaks fats into glycerol and fatty acids) into the small intestine where most digestion and absorption takes place. Conclusion Most over-the counter antacid medications combat the normal stomach acid (HCl) to produce a more nearly neutral pH (closer to a pH of 7), and as the pH rises above 4, pepsin activity decreases or stops. Additionally, it has been observed that because our bodies are designed to maintain a normal, constant internal environmental balance – homeostasis – typically, consumption of antacids results in the stomach actually secreting more hydrochloric acid to try to compensate for the imbalance caused by the antacids, until the organ because sluggish by over usage . Thus, while consumption of antacids in rare circumstances may be helpful to soothe gastric ulcer H pylori, this application of alkaline substances with help the colony grow damaging more of the lining of the stomah. The quick fix of an antacid might not fit into your long term health plans. |
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